Quote from CosmonautFirst of, please stop grouping all drugs with marijuana, as you seem to be doing. Now I'm going to assume you're talking about marijuana ok, so that's what I'm going to argue against.
No, consumers do not deny that it can take control of yourself, the Surgeon General of the United States does. And consumers of course heh.
Now, every thing can cause dependence; coffee, sugar, pizza what have you. The important thing is if it has chemicals that CAUSES these dependence, marijuana has none. SO if someone was to get "addicted" to marijuana, they would also have the same chances as getting addicted to coffee, sugar pizza what have you. It has nothing to do with the actual substance, but the person who takes it.
There is no evidence that provides any proof that marijuana harms you in any way, unless of course you smoke it but that has nothing to do with the substance. That's just common sense if you inhale smoke, it isn't good for you. There is nothing in the plant itself that harms you, and before you say no it does not kill brain cells that is just a stupid myth.
Quote from KaasI'm kinda annoyed of the always saying "marijuana is not addictive"...
There are people that are socially messed up because of weed, be it addiction or trigged mental-disorder. I've met them, my dad works with them. So big whoop for the next article you read saying it's not harmful, for some people it is -_-.
Don't you just love it when a thread loses it's purpose?
Quote from CosmonautYeah, I know it is for some people I said that didn't I? There isn't, however a chemical in marijuana that makes it addictive like meth or ciggs though. That's what my point was. It's all mental.
Well I didn't like the threads purpose in the first place so i don't mind realy. :D
Quote from KaasI'm kinda annoyed of the always saying "marijuana is not addictive"...
There are people that are socially messed up because of weed, be it addiction or trigged mental-disorder. I've met them, my dad works with them. So big whoop for the next article you read saying it's not harmful, for some people it is -_-.
Don't you just love it when a thread loses it's purpose?
Quote from notmaggotyeah but you could say that about anything. replace "marijuana" with "chocolate" or "cake" and "socially messed up" with "fat".
Quote from KaasWell, mental it may be, but marijuana triggers a lot of laying-low defects more quickly then shrooms' or let's say LSD. I met this one dude who, if he doesn't take his medicine, thinks "they are coming out of the electrical sockets", he totally trips...
Quote from schizophrenia.comProfessor John Henry, clinical toxicologist at Imperial College London said research has shown that people with a certain genetic makeup who use the drug face a ten times (1000%) higher risk of schizophrenia. (for example - if your risk of schizophrenia was 6% (due to a family history of mental illness) prior to taking cannabis, it could be 60% -- or more likely than not - after taking cannabis). Every person is different (i.e. has different genes and different environments) - so this "10 Times Higher Risk with cannabis use"- is just a generalization, and it may or may not apply to a given person.
The increased risk applies to people who inherit variants of a gene named COMT and who smoked cannabis as teenagers. About a quarter of the population have this genetic make-up and up to 15 per cent of the group are likely to develop psychotic conditions if exposed to the drug early in life. Neither the drug nor the gene raises the risk of psychosis by itself.
Quote from KaasWell, mental it may be, but marijuana triggers a lot of laying-low defects more quickly then shrooms' or let's say LSD. I met this one dude who, if he doesn't take his medicine, thinks "they are coming out of the electrical sockets", he totally trips...
The scientists say the drug only seems to affect people who are genetically predisposed to getting schizophrenia (meaning they will get it anyway). As schizophrenia manifests itself during adolescence, and many people start taking cannabis during adolescence - it is just coincidence that some people develop the mental illness soon after they start taking the drug.
According to a new report by a British government advisory body, the regular use of cannabis though it can have real and significant mental health effects it is unlikely to cause schizophrenia.
The endogenous cannabinoid system has recently been shown of particular importance in the pathophysiology of acute schizophrenia. It interacts with various neurotransmitter systems in the central nervous system including the dopaminergic, glutamatergic and GABAergic system. While the psychedelic properties of the natural cannabis compound delta-9-tetrahydrocannabinol are widely known, there is some experimental and clinical evidence that other herbal cannabinoid compounds may have antipsychotic properties.
Based on these confounders we designed a four week, double-blind, controlled clinical trial on the effects of purified cannabidiol, a major compound of herbal cannabis, in acute schizophrenia and schizophreniform psychosis compared to the antipsychotic amisulpride. The antipsychotic properties of both drugs were the primary target of the study. Furthermore, side-effects and anxiolytic capabilities of both treatment strategies were investigated.
Cannabidiol significantly reduced psychopathological symptoms of acute psychosis after both, week two and four, when compared to the initial status. There was no statistical difference of this effect to the control condition. In contrast, Cannabidiol revealed significantly less side effects when compared to amisulpride.
This phase II clinical trial on the effects of Cannabidiol in acute schizophrenia and schizophreniform psychosis raises evidence for its antipsychotic properties that exceeds by far the evidence from open observations available up to now. Furthermore, it raises evidence that the endogenous cannabinoid system may provide a valid target in the search for new treatments for schizophrenia.
Acknowledgements: Funded by the Stanley Medical Research Institute (00-093 to FML) and the Koeln Fortune Program (107/2000 + 101/2001 to FML).
Besides identifying antibacterial capability, the researchers wanted to figure out why these cannabinoids are so good at killing bacteria. They obviously are very effective at specifically targeting some vital process in the bacteria. Unfortunately, even after extensive work at modifying the cannabinoids and comparing their activities, that targeting mechanism remains a mystery. The scientists were able to figure out that the position of the n-pentyl chain (orange) relative to the terpenoid moiety (blue) serves to control lipid affinity.